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This massive genomic footprint on the nervous system suggests that GHK-Cu acts as an epigenetic modifier, resetting pathological gene expression patterns toward healthier states

Antibodies used were as follows: FoxO3a (Cell Signaling Technology, #12829), phospho-FoxO3a (Ser253) (HUABIO, ST49-01), Atrogin-1 (Proteintech, 67172-1-Ig, RRID: AB_2882468), MuRF-1 (Proteintech, 55456-1-AP, RRID: AB_11182630), GAPDH (Proteintech, 60004-1-Ig, RRID: AB_2107436), and HRP-conjugated secondary antibodies (Proteintech, SA00001-1/SA00001-2)

Analysis of relevant literature along with the results from this study helped us to establish the hypothesis that a low GSH/iron in the blood plasma and serum respectively may serve as potential biomarkers for the risk of developing AD