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To explore the relevance of GSH to the distinct redox properties of FoB and MZB in vivo, we generated B cell-specific Gclc -deficient mice by crossing Gclc fl/fl mice with Mb1-Cre + mice ( Gclc fl/fl Mb1-Cre + ), in which the Cre recombinase gene is expressed under the control of the B cell-specific promoter Mb1 46

In vivo , FTO knockdown significantly elevates YTHDF2 expression in the striatum of PD mice, inhibits m 6 A demethylation of BAP1 and reduces BAP1 expression, upregulates SLC7A11 expression, decreases -Syn, MDA, Fe 2+ , and 4-HNE levels, suppresses neuronal ferroptosis and disruptions in the amino acid antioxidant system, and promotes dopaminergic neuronal survival in PD mice (Li et al., 2025c)

LWT 169:113981

2025;328(2):C710C727