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Neurological Relevance Preclinical evidence suggests broader neuroprotective potential: In traumatic brain injury models, SS-31 reduced mitochondrial swelling, neuronal loss, and neuroinflammation Aged mice treated with SS-31 showed significantly improved exercise tolerance, muscle strength, and mitochondrial efficiency Studies demonstrate protection against ischemic injury and enhanced cardiac output Phase 3 trials are ongoing for dry age-related macular degeneration and primary mitochondrial myopathy (Stealth BioTherapeutics pipeline) Implications for Brain Aging Mitochondrial dysfunction is a hallmark of brain aging and neurodegeneration

This receptor specificity reduces stimulation of other pituitary axes that regulate cortisol, prolactin, or ACTH secretion

You then have to reassemble it intracellularly, which partially defeats the purpose of supplementing the completed molecule

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Apolipoprotein E isoform-dependent effects on anxiety and cognition in female TR mice