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Unlike sepsis, where treatments are primarily directed against LPS-induced inflammatory responses (e.g., anti-endotoxin antibodies), SAP-induced glycocalyx damage is predominantly driven by pancreatic enzyme activation (e.g., trypsin) and intestinal endotoxin release

Group mean metabolite concentrations were significantly lower in the patients than in the controls for both glutathione and glutamine in the ACC, and significantly lower for all three metabolites in the ACC in the subset of patients with residual schizophrenia

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doi: 10.1038/cmi.2014.89 215 PedreB.BarayeuU.EzeriaD.DickT

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