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Elevated histone acetylation was confirmed in an additional cell line and shown to decrease through the depletion of KG or the suppression of KGDDs using knockdown or inhibition of IDH1, glutamine starvation or succinate supplementation (Fig

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glutathione S-transferases (GST), which catalyse the conjugation of GSH to electrophilic and xenobiotic compounds for detoxification and excretion

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This is now considered a general paradigm for signaling by many transmembrane receptors