Studies discovered mitochondrial-derived peptide called MOTS-c has been shown to significantly reduce the level of pro-inflammatory factors in mice and increase anti-inflammatory factors and insulin-stimulated glucose treatment rates, as well as glucose homeostasis (79)
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In animal models, GHK-based nanoparticle formulas achieved 9496% wound closure by day 11, along with thicker new tissue, more collagen, and lower inflammation markers. It's a good example of how researchers are trying to get more out of the same peptide by rethinking how it's delivered
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