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Furthermore, the resulting chlorinative stress may lead to multiorgan damage in COVID-19 through various mechanisms, including oxygen competition for heme-binding sites, decreased oxygen saturation, hemoglobin-heme iron oxidation, heme degradation, and iron release [88]

then reinstating extracellular calcium at 2 mM induced a large increase in intracellular Ca2+, reflecting the SOCE (Figure 2C)

A critical axis in this balance is between pro-inflammatory T helper 17 (Th17) cells and anti-inflammatory regulatory T (Treg) cells ( However, many existing reviews have primarily catalogued these metabolic differences in isolation

PubMed Sikiric P, Seiwerth S, Rucman R, Kolenc D, Vuletic LB, Drmic D, et al

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