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However, the number of these newly formed neurons is extremely low, at least in epilepsy, and a therapy focused on neuronal restitution does not appear to be sufficient so far (Parent et al., 1997)

Glucagon-like peptide-1 receptor regulates endoplasmic reticulum stress-induced apoptosis and the associated inflammatory response in chondrocytes and the progression of osteoarthritis in rat

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TB-500 provides complementary anti-inflammatory activity through multiple mechanisms including direct inhibition of NF-B signaling, modulation of immune cell phenotypes, and promotion of inflammatory resolution